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IGF and proinsulin share homology such that both of their respective receptors, IGFR and the insulin receptor (IR), will bind to the alternate growth factors albeit with reduced affinity (95)

This lowers the production of glutathione, which requires cysteine rather than cystine, and now there is a vicious circle mechanism that preserves this malfunction and keeps you sick That's the basic biochemical mechanism of CFS everything else flows from this Here's how I believe the fatigue occurs: The cells have little powerplants in them, called mitochondria

114 WuZ.GengY.LuX.ShiY.WuG.ZhangM.et al (2019)