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This means that the synapse mitochondria have additional functions at the nerve terminals, which may include Ca 2+ buffering directly affecting the firing probability of neurons [37, 38]

height: Each member of the treatment groups received an icv injection of scopolamine hydrobromide (70 nmof in 2 aCSF over a duration of 20 s) 20 min prior to testing followed by Dihexa (300 pmol in 2 aCSF), Hinge (300 pmol in 2 aCSF), or Hinge + Dihexa (300 pmol in 4 aCSF) 5 min prior to testing

Glutathione IV therapy is a time-efficient solution

Collectively, these recent contributions argue that future GSH-centric redox sensing and nanomedical approaches should (i) incorporate organelle targeting (especially mitochondria) to exploit subcellular redox heterogeneity, (ii) embed analyte-responsive activation into both therapeutic and imaging components, (iii) combine biochemical sensing with optical or thermal engineering for spatiotemporal control, and (iv) favor clinically translatable carriers (e.g., PLGA) for delivery