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ISO-administered rats showed increased CK-MB activity in the plasma, consistent with previous reports 38,39

The mutated ATXN3 gene is translated into an abnormal polyglutamine (polyQ) tract within the ataxin-3 (ATXN3) protein, which has normal expression levels even in the presence of this mutation [5]

Recognizing this duality highlights the need for a comprehensive approach that balances symptom relief with potential impacts on gut health, ensuring that interventions contribute positively to the multifactorial nature of ME/CFS [7]

Females typically have a wider pelvis, greater Q-angle, and more significant ligamentous laxity compared to males