Its primary mechanisms include promoting angiogenesis (new blood vessel formation) through VEGF upregulation, modulating the nitric oxide system in a context-dependent manner, activating the FAK-paxillin pathway to drive cell migration and tissue rebuilding, and regulating multiple growth factors (PDGF, TGF-beta, FGF) involved in organized tissue repair
The enhanced fibroblast migration via FAK-paxillin pathway activation brings repair cells to injured tendons more rapidly
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If a serious complication arose (say, an immune reaction or a cancer diagnosed down the line), the patient might argue they werent properly warned because the doctor themselves could not know the risks