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A.ThomasG

As a result, W276 6.48 toggles to further induce significant displacement of F272 6.44 and the cytoplasmic segment of TM6, resulting in the activation of GHSR (Fig

Hyperphosphatemia induces senescence in human endothelial cells by increasing endothelin-1 production

Mechanistic study has shown that PGC-1 plays an important role in the modulation of the UPR through coactivating ATF6, a well-characterized sensor in UPR, thus contributing to skeletal muscle adapt to exercise training