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We show that depleting macrophages of 99% of GSH does not exacerbate the inflammatory gene expression profile in the RAW264 macrophage cell line or increase expression of inflammatory cytokines in response to the toll-like receptor 4 (TLR4) agonist lipopolysaccharide (LPS)

Mechanisms: MT-I primarily activates the MC1R, while MT-II has increased affinity towards the MC3R and MC4R [6, 9]