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This c-MET mutant showed deficiency in HGF-dependent activation (Fig

Mechanism of Action (Research Context) Modulation of cellular signaling and molecular communication pathways Enhancement of growth factor sensitivity in in-vitro models Regulation of gene expression and transcriptional activity Influence on mitochondrial ATP production and energy metabolism Support of enzymatic activation and peptide-mediated remodeling processes Primary Research Applications Regenerative Cell Biology: Investigating tissue repair, angiogenesis, and cellular regeneration models

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mEPSC amplitude distributions were similarly diminished by acivicin, and more so by MeAIB, although combinations of acivicin and MeAIB did not further impair the effect of MeAIB alone (Fig