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PNM as a luminal filler for inert silicone conduit was shown to produce gait, electrophysiologic, and histologic outcomes equivalent to autografting for subcritical nerve gap injuries over or at 24 weeks in a rat model

At the molecular level, the mechanism has been structurally resolved by NMR spectroscopy (Nature Communications, 2025): FOXO4-DRI binds to the disordered p53 TAD2 domain and forms a transiently folded complex through both the FOXO4-derived peptide region and its cationic cell permeability element

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Lets dive into what the research suggests and what our experience has shown us