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Conclusion While mostly combined with Semaglutide, Cagrilintide still offers plenty of benefits

The aim of this review is to explore the key role of mitochondrial dysfunction in T cell exhaustion and its mechanisms, to summarize the strategies for targeting mitochondria to restore the function of T ex cells, and to provide a theoretical basis for the development of novel mitochondria-targeted therapies

Interestingly, recent studies have proposed that DNA methylation can lead to the exclusion of a histone variant (H2A.Z), preferentially located at active gene promoter 36,37

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