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The phenomenon of cellular senescence in cancer cells triggered by radiotherapy or chemotherapy is termed therapy-induced senescence (TIS) [54]

& Stadtman, E

These findings align well with our correlation analyses, in which butyrate levels positively correlated with muscle mass and function, while showing negative associations with the oxidative stress marker MDA and atrophy markers Atrogin-1 and MuRF-1

This broad regulatory activity includes upregulation of tissue repair genes, collagen synthesis pathways, antioxidant defense systems, and anti-inflammatory signaling networks, alongside simultaneous downregulation of genes associated with tissue destruction, inflammation, and fibrosis