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For instance, hypoxia can drive the transformation of macrophages into the M1 pro-inflammatory phenotype by stabilizing hypoxia-inducible factor-1 (HIF-1), promote glycolytic reprogramming of activated naive B cells and naive T cells, and simultaneously inhibit the immunosuppressive function of regulatory T cells (Tregs) thereby triggering and amplifying inflammatory responses

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IGF-1 DES has 20-30 minute half-life, minimal IGFBP binding, highest receptor affinity and potency, localized effects at injection site, and requires frequent dosing for sustained effects