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Cysteine residues in Keap1 act as sensors for electrophiles and oxidants, and their oxidation leads to the release and subsequent translocation of Nrf2 into the nucleus where it can bind to the ARE4 sequence in the promoter region of genes involved in antioxidant defense (Moinova and Mulcahy 1999)

Up to 40% of hypothyroid patients may have low stomach acid [57, 58]

Instead of cells dying to be replaced by new, healthy cells, the affected cells simply continue to divide and multiply, causing a cluster of cells that pack together and form a lump of tissue

The gap between gene data and clinical outcomes requires bridging through human trials