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2a,b), consistent with ABHD11 regulating lipoyl glutarylation rather than lysine glutarylation

Inhibition of glutathione markedly enhanced H 2 O 2 - or ARS-induced cytotoxicity in HeLa and A549 cell lines but modestly enhanced the cytotoxicity of H 2 O 2 and even did not affect the cytotoxicity of ARS in HepG2 cells

This is why **N-acetyl-L-cysteineNACis so important.** NAC provides the body with a usable precursor to cysteine, giving liver cells and other cells the raw material they need to manufacture their own glutathione where it is actually required