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In these circumstances, the activation of NrF2 and tHO-1 at the nuclear level is actively used by cancer cells as a protective system against oxidative and ER stress to sustain cell survival and the acquisition of a more aggressive tumour phenotype

Like, what have you seen short term, long term, because you, it sounds like you guys have been working with us for a long time, in the response of people's bodies and conditions

5a) The source of ROS and the mechanism by which these ROS drive possibly drive this proinflammatory phenotype therefore warrant further investigation