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FIS1-recruited TBC1D15 drives Rab7 GTP hydrolysis, promoting untethering of mitochondria and lysosomes [64] and leading to DRP1-mediated separation of the peripheral mitochondrial regions [36]

These efforts are complicated by the fact that the same antibody mechanisms that are often proposed to result in ADE of infection are responsible for protection from viral disease in vivo

FIGURE 1 2.2 GSHs role in brain-resident immune cells In the CNS, GSH protects mitochondrial function, prevents lipid peroxidation, and maintains the redox state of cysteine residues in proteins

For instance, apoptosis can be triggered by inflammatory signals or metabolic stress