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These are often the injuries that frustrate patients because they are painful enough to limit activity but not always severe enough to trigger aggressive treatment in the traditional medical system

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TBARS, thiobarbituric acid-reactive species

Key contributors to atherogenesis include oxidative stress (excess ROS oxidizing lipids and damaging endothelial cells), vascular inflammation (cytokine and chemokine release recruiting more immune cells), endothelial dysfunction (reduced NO bioavailability and loss of barrier integrity), and dyslipidemia (elevated LDL/triglycerides and low HDL)