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Mitochondria are central to the regulation of apoptosis, where pro-apoptotic signals trigger OMM permeabilization, releasing pro-apoptotic factors like cytochrome c and activating caspases

The DNA-dependent protein kinase catalytic subunit promotes sepsis-induced cardiac dysfunction through disrupting INF-2-dependent mitochondrial dynamics

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Our exploration of cutting-edge methods, such as causal inference and multi-omics fusion, may be insufficient