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However, it remains to be determined whether its beneficial effects are primarily mediated via the inhibition of direct PPAR target genes, such as CD36, or through the PPAR-mediated repression of proinflammatory transcription factors, such as NF-kB

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Mossa ATH, Swelam ES, Mohafrash SMM (2015) Sub-chronic exposure to fipronil induced oxidative stress, biochemical and histotopathological changes in the liver and kidney of male albino rats