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More recent studies further extend its protective profile by demonstrating that salidroside limits regulated cell death pathways, including ferroptosis, pyroptosis, and PANoptosis, through coordinated regulation of PI3K/AKT, Nrf2-dependent antioxidant signaling, and hypoxia-responsive pathways, thereby preserving tubular and microvascular integrity in ischemic, septic, and crystal-associated AKI models (Tang et al

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Specifically, the substitutions at the 2nd, 15th, and 27th positions may help the peptide resist being broken down by enzymes and extend its half-life